启动子 ATP依赖的染色质重塑复合物与肺癌研究进展(7)
[17] Hoffman G R,Rahal R,Buxton F,et al.Functional epigenetics approach identifies BRM/SMARCA2 as a critical synthetic lethal target in BRG1-deficient cancers[J].Proc Natl Acad Sci USA,2014,111(8):3128-3133.
[18] Oike T,Ogiwara H,Tominaga Y,et al.A synthetic lethality-based strategy to treat cancers harboring a genetic deficiency in the chromatin remodeling factor BRG1[J].Cancer Res,2013,73(17):5508-5518.
[19] Kozmik Z,Machon O,Kralova J,et al.Characterization of mammalian orthologues of the Drosophila osa gene:cDNA cloning,expression,chromosomal localization,and direct physical interaction with Brahma chromatin-remodeling complex[J].Genomics,2001,73(2):140-148.
[20] Huang J,Zhao Y L,Li Y,et al.Genomic and functional evidence for an ARID1A tumor suppressor role[J].Genes Chromosomes Cancer,2007,46(8):745-750.
[21] Zhang Y,Xu X,Zhang M,et al.ARID1A is downregulated in non-small cell lung cancer and regulates cell proliferation and apoptosis[J].Tumour Biol,2014,35(6):5701-5707.
[22] Manceau G,Letouze E,Guichard C,et al.Recurrent inactivating mutations of ARID2 in non-small cell lung carcinoma[J].Int J Cancer,2013,132(9):2217-2221.
[23] Papadakis A I,Sun C,Knijnenburg T A,et al.SMARCE1 suppresses EGFR expression and controls responses to MET and ALK inhibitors in lung cancer[J].Cell Res,2015,25(4):445-458.
[24] Zhang X,Fu L,Xue D,et al.Overexpression of Rsf-1 correlates with poor survival and promotes invasion in non-small cell lung cancer[J].Virchows Arch,2017,470(5):553-560.
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